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* v ' : 5S*'J ' *i,'~ r•>** :-v^-*' - *:\:'*v j .'-' -> ->T ."•« -'* •->_- ''*? "v.7r *d baetoria or similar injurious substances gala access So liTia* Siawoa iho local capillary bed reepOnds sad. permits the escape of fluid and leucocytes into tha soaa of injury. Most of the extiUTSSated fluid returns to tho circulation by the lynphatie* beeanse the inflaanatoiy edeo* eoapresses capillar!** and. venules Vat distands the lymphatics, which arc anchored ly amtfetti septa to the interstitial fluid compartment, fha aodified interstitial fluid frou the injured area, trhlch. returns Tin traverses lynphold tissue In the lymsh. nodes, ft* lymphoid tissue, vlthia 3 to 7 days* responds Vy liberating antibodies ddth *nt«r the oir^ eulntion and return to the injured site where they eerwe to neutralise the Injurious agent by uniting antibody with antigeaicnosbabsneeo. It is customary to describe the immune response which follows the inflaanntory reaction to local injury as a co-defensive mechanism, ehlph is M gMy specific and nearly 100 per cent efficient in its end results, A study of the allergic diseases such as hay fewer, asthma, urticaria and ecsema f.r%A the occurrence of each drsnatic reactions as fatal anaphylaxis or 6Xten— eive necrosis in the Arthaa 1 * phenoaenon indicates that antibody-antigen reaction is not aa accurate aad efficient as immunologists lead US to bolievo* * Supported by Grant [CTT3J-Tr^